Thanks. Indeed, selfishly (I wouldnt be bothing if I didn't have lipoma), it is my intention to try and decypher possible good/bad/indifferent treatment/cure information. To do so though (to stand any chance realistically) I have felt it necassary to cast the net wider to understand some basic medical terms, facts and principals surrounding normal adipose construction and, given the pathology similarities, that of lipoma. In this regard, I started another topic recently:
'what exactly is a lipoma?
I am interested in what you say with regards to cell division. Let's see if I can get this right. I have the 12q mutation and have a proposition to lipoma as we have discussed. For the vast majority of my body, I have normally described and functioning adipose (allbeit with the mutation). However, I have approx 50 instances of uncontrollable cell division and growth within my adipose with a result of lipoma tumour. Now, if I take it that I have lost the normal control of my bodies cell activity at these points (inc. division and growth), then something different or event on
must have occurred here, trigger, to make this happen (ie. preposition + trigger = lipoma). We dont know what event has happened, BUT the resultant pathology differences might provide some clues as to WHY and perhaps also to afford an event off
. For instance, there are a number of reports that show irregular enzyme activities within lipoma - Lipoprotein Lipase (LPL) and Hormone Sensitive Lipase (HSL). One report states "It is likely that the high lipoprotein lipase activity of lipoma contributes to the growth of the tumour". This begs the question, are these resultant or attributed or even indeed linked to event on
, and could they be controlled to create a treatment. What else is out there that might contibute similarly?